Which one of these is not a type of transporter protein
Which one of these is not a type of transporter protein
Which one of these is not a type of transporter protein
Questions
Which оne оf these is nоt а type of trаnsporter protein
Mоdule 4 Optiоn A A scientist is investigаting the effects оf а newly discovered mаrine toxin called Neurotoxin-X, which is found in certain tropical fish. This toxin selectively blocks voltage-gated sodium (Na⁺) channels, preventing them from opening. People who consume contaminated fish experience muscle paralysis, numbness, and difficulty breathing, suggesting that the toxin interferes with normal nerve signaling. Explain how a neuron at rest maintains its resting membrane potential. In your response, describe the role of the sodium-potassium pump and leakage channels in maintaining the charge difference across the membrane. Describe the sequence of events in a normal action potential, including the roles of voltage-gated channels in depolarization, repolarization, hyperpolarization, and the refractory period. How would a toxin that blocks voltage-gated sodium channels affect action potential generation? Explain why neurons exposed to Neurotoxin-X would be unable to communicate with muscles or other neurons. How could the effects of Neurotoxin-X impact nerve signal conduction? Option B A pharmaceutical researcher is testing a new drug, NeuroBlock, designed to treat chronic pain by blocking voltage-gated potassium (K⁺) channels in neurons. During clinical trials, some participants report prolonged muscle contractions, twitching, and difficulty relaxing their muscles after movement. The researcher suspects that the drug is interfering with normal action potential repolarization and disrupting nerve signaling. Explain how a neuron at rest maintains its resting membrane potential. In your response, describe the role of the sodium-potassium pump and leakage channels in maintaining the charge difference across the membrane. Describe the sequence of events in a normal action potential, including the roles of voltage-gated channels in depolarization, repolarization, hyperpolarization, and the refractory period. How would blocking voltage-gated potassium channels affect action potential generation and recovery? Explain why neurons exposed to NeuroBlock might cause prolonged muscle contractions and difficulty relaxing. How could the effects of NeuroBlock impact nerve signal conduction?